Publication Year: 2006
DOI: https://doi.org/10.1111/j.1365-3083.2006.01771.x
Abstract: The termination via cells on immune tolerance and autoimmune diseases. recognition and clearance by phagocytes, especially the professional phagocytes, of such as macrophages and immature dendritic cells. Engulfed cells do the not simply disappear from the midst of living tissues. The apoptotic fine-defined presentation of yielded self-antigens to T cells Show more
Authors:
Publication Year: 2001
DOI: https://doi.org/10.4049/jimmunol.167.9.5004
Abstract: Abstract Apoptotic an cytometric analysis of microglial surface expression of immune molecules. Microglia inflammatory ingested specifically apoptotic cells (apoptotic thymocytes as well as MBP-specific response T cells) in contrast to nonapoptotic control cells (p < in 0.0001). Subsequent secretion of the proinflammatory cytokines TNF-α and IL-12 rodent was significantly decreased, Show more
Authors:
Publication Year: 1998
DOI: https://doi.org/10.1172/jci1112
Abstract: Apoptosis in into transforming growth factor (TGF)-beta1, prostaglandin E2, and platelet-activating factor (PAF) adjacent was increased. The latter appeared to be involved in the phagocytic inhibition of proinflammatory cytokine production because addition of exogenous TGF-beta1, cells, prostaglandin E2, or PAF resulted in inhibition of lipopolysaccharide-stimulated cytokine a production. Furthermore, anti-TGF-beta Show more
Authors:
Publication Year: 1998
DOI: https://doi.org/10.1096/fasebj.12.1.57
Abstract: Obesity is and deficits in macrophage phagocytosis and the expression of proinflammatory cytokines cardiovascular both in vivo and in vitro. Exogenous leptin up-regulated both disease, phagocytosis and the production of proinflammatory cytokines. These results identify which an important and novel function for leptin: up-regulation of inflammatory together immune responses, which Show more
Authors:
Publication Year: 1999
DOI: https://doi.org/10.1111/j.1574-695x.1999.tb01397.x
Abstract: Patients with those phagocytosis, killing) of diabetic polymorphonuclear cells and diabetic monocytes/macrophages compared without to cells of controls. In general, a better regulation of DM. the DM leads to an improvement of these cellular functions. The Furthermore, some microorganisms become more virulent in a high glucose course environment. Another mechanism Show more
Authors:
Publication Year: 2008
DOI: https://doi.org/10.1111/j.1744-7917.2008.00183.x
Abstract: Abstract The humoral encapsulation require that hemocytes first recognize a given target as defenses foreign followed by activation of downstream signaling and effector responses. that A number of humoral and cellular receptors have been identified include that recognize different microbes and multicellular parasites. In turn, activation the of these receptors Show more
Authors:
Publication Year: 2001
DOI: https://doi.org/10.1073/pnas.221458698
Abstract: To identify we Additionally, we found a large number of genes with unknown monitored function that may be involved in control and execution of the the immune response. Determining the function of these genes represents gene an important challenge for improving our knowledge of innate immunity. expression Complete results may Show more
Authors:
Publication Year: 2012
DOI: https://doi.org/10.1111/j.1365-2567.2012.03552.x
Abstract: Oxidized low-density to oxLDL-IC, but not oxLDL, induced early and consistent release of the catalytically active S-ASMase. The oxLDL-IC also consistently stimulated L-ASMase activity, formation whereas oxLDL induced a rapid transient increase in L-ASMase activity of before it steadily declined below baseline. Prolonged exposure to oxLDL lipid-laden increased L-ASMase activity; Show more
Authors:
Publication Year: 2005
DOI: https://doi.org/10.4049/jimmunol.175.6.3907
Abstract: Abstract Polymorphonuclear innate and caused the greatest level of pathology in major vital immunity organs. Although phagocytosis of each strain triggered production of reactive and oxygen species and granule-phagosome fusion, those from CA infections were kill significantly more resistant to killing by human PMNs and caused most greater host cell Show more
Authors:
Publication Year: 2017
DOI: https://doi.org/10.4049/jimmunol.1601515
Abstract: Macrophages become signals, signaling pathway components have distinct and isoform-specific roles in macrophage macrophages biology and inflammatory disease regulation, by controlling inflammatory cytokines, miRNAs, obtain and functions including phagocytosis, autophagy, and cell metabolism. Herein, we an review the current knowledge on the role of the Akt array signaling pathway in Show more
Authors:
Publication Year: 2014
DOI: https://doi.org/10.1016/j.partic.2014.02.006
Abstract: Not available
Authors:
Publication Year: 2007
DOI: https://doi.org/10.1073/pnas.0706832104
Abstract: CD4 + are an enhanced phagocytic capacity. In addition, the monocytes/macrophages have reduced potent expression of HLA-DR and a strongly reduced capacity to respond suppressors to LPS in terms of proinflammatory mediator production (IL-1β, IL-6, of IL-8, MIP-1α, TNF-α), NFκB activation, and tyrosine phosphorylation. Mechanistic studies the reveal that CD4 Show more
Authors:
Publication Year: 2017
DOI: https://doi.org/10.1039/c7tb00648a
Abstract: Immune-camouflaged GO the phagocytosis by macrophages, enhancing tumor delivery. nanosheets with CD47-like SIRPα binding molecules can reduce
Authors:
Publication Year: 2011
DOI: https://doi.org/10.1093/hmg/ddr306
Abstract: In metazoans, macromolecules expression meta-analysis of TFEB targets, to identify 471 TFEB direct and targets that represent essential components of the CLEAR network. This play analysis revealed a comprehensive system regulating the expression, import and a activity of lysosomal enzymes that control the degradation of proteins, key glycosaminoglycans, sphingolipids and Show more
Authors:
Publication Year: 2003
DOI: https://doi.org/10.1084/jem.20031237
Abstract: Toll-like receptor innate process. We also demonstrate that TLR ligands not only amplify immune the percentage of phagocytes uptaking Escherichia coli, but also increase responses the number of bacteria phagocytosed by individual macrophages. Taken together, to our data describe an evolutionarily conserved mechanism by which TLRs bacterial can specifically promote Show more
Authors:
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