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'referenced_works_count': 0, 'referenced_works': [], 'related_works': [], 'abstract_inverted_index': {'世袭出血性的毛细管扩张(HHT': [0], ')': [1, 12, 26, 63], '是在': [2], '5000': [3], '~': [4], '8000': [5], '个人发生在约一的稀罕': [6], 'autosomal-dominantly': [7], '继承的疾病。当一个人介绍下列四个标准中的三个时,': [8], 'HHT': [9, 49], '的临床的诊断被做:在大脑的家庭历史,周期性的鼻血,粘膜与皮肤的': [10], 'telangiectasis,和动静脉的畸形性(AVM': [11], ',肺,肝并且胃肠(官方补给)': [13], '道。尽管': [14], 'epistaxis': [15, 65], '是最普通的介绍症状,影响肺,大脑和官方补给的道的': [16], 'AVM': [17], '挑起更严肃的结果。在': [18], 'endoglin': [19], '的异质接合的变化,': [20], 'activin': [21], '像受体的': [22], 'kinase': [23], '1': [24], '(ACVRL1;ALK1': [25], ',并且': [27], 'SMAD4,涉及转变生长因素的基因': [28], '--': [29], '家庭发信号串联,原因': [30], 'HHT。我们这里报导介绍了': [31], 'melena': [32], '和官方补给的流血事件的': [33], '63': [34], '74': [35], '岁的男病人的案例,证明被从多重胃的': [36], 'angiodysplasia': [37, 41], '流血引起。Esophagogastroduodenoscopy': [38], '在整个胃揭示了多重': [39], 'angiodysplasia。内视镜的氩血浆凝结被执行控制从胃的': [40], '流血。病人与': [42], 'hemoptysis': [43], '和': [44], 'hematochezia': [45], '的事件若干次被招收。一年以前,病人由于右边的软弱被就医,它被左基础': [46], 'ganglia': [47], '出血作为': [48], '表示的部分引起。家庭历史上,病人母亲和大姐死了,由于': [50], 'intracranial': [51], '出血,并且他的长子被受不了为': [52], '20': [53], '年的周期性的': [54], 'epistaxis。基因研究在': [55], 'exon': [56], '揭示了一个变化': [57], '3': [58], 'ALK1': [59], '(c.199C': [60], '>': [61], 'T;p.Arg67Trp': [62], '在介绍': [64], '的': [66], 'proband': [67], '和他的长子。': [68]}, 'cited_by_api_url': 'https://api.openalex.org/works?filter=cites:W3149544334', 'counts_by_year': [{'year': 2012, 'cited_by_count': 1}], 'updated_date': '2024-09-20T00:37:46.843234', 'created_date': '2021-04-13'}