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'mediated': [56, 66], 'alternative': [58], 'lengthening': [59], 'telomeres': [61], 'anti-apoptotic': [64], 'response': [65], 'signaling': [70, 74, 97], 'autocrine': [73], 'leading': [75], 'activation': [77], 'PI3K/AKT': [80], 'pathway.': [81], 'In': [82], 'presence': [84], 'KS': [87, 117], 'growth': [88], 'factor': [89], 'VEGF,': [90], 'this': [91, 126], 'mechanism': [92], 'can': [93, 106], 'sustain': [94], 'suppression': [95], 'immortalizing': [100, 110], 'gene.': [101], 'We': [102], 'conclude': [103], 'cause': [107], 'oncogenic': [109], 'event': [111], 'recapitulate': [113], 'aspects': [114], 'angiogenic': [118], 'phenotype': [119], 'cells,': [123], 'pointing': [124], 'gene': [127], 'as': [128], 'a': [129], 'pathogenic': [130], 'determinant': [131], 'KSHV.': [133]}, 'cited_by_api_url': 'https://api.openalex.org/works?filter=cites:W2044627970', 'counts_by_year': [{'year': 2024, 'cited_by_count': 2}, {'year': 2023, 'cited_by_count': 3}, {'year': 2022, 'cited_by_count': 2}, {'year': 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