Title: Constrictive Remodeling: Do Concepts of Remodeling During Chronic Hypertension Apply to Restenosis?
Abstract: Restenosis following angioplasty traditionally has been attributed to intimai hyperplasia involving the release of growth factors. Growth factors presumably induce a subgroup of smooth muscle cells in the media of the vessel wall to undergo phenotypic transformation, migrate into the intima, proliferate and secrete extracellular matrix [1]. A new concept that has been proposed recently is that constrictive remodeling with a reduction in vessel caliber may be another mechanism that contributes to restenosis [2, 3].
Publication Year: 1997
Publication Date: 1997-01-01
Language: en
Type: book-chapter
Indexed In: ['crossref']
Access and Citation
AI Researcher Chatbot
Get quick answers to your questions about the article from our AI researcher chatbot