Abstract: Ascorbic acid deficiency has been traditionally recognized by the characteristic (scorbutic) signs and symptoms of clinical scurvy, which respond to (i.e., can be cured by) rich dietary sources of vitamin C such as citrus fruits or a dietary vitamin C supplement. Milder deficiency results in low or negligible concentrations of vitamin C in serum, plasma, and many tissues without causing overt clinical abnormality. Vitamin C occurs in several forms, of which the fully reduced form, ascorbic acid, is responsible for a variety of redox roles in the body, for example, in certain key enzymes and reactions. An intake of 10 mg day−1 in adult humans can cure and prevent clinical scurvy, but higher intakes are required to replete body stores and optimize functionality.
Publication Year: 2013
Publication Date: 2013-01-01
Language: en
Type: book-chapter
Indexed In: ['crossref']
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Cited By Count: 5
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