Publication Year: 1982
DOI: https://doi.org/10.1016/0006-8993(82)91048-4
Abstract: Not available
Authors:
Publication Year: 1988
DOI: https://doi.org/10.1073/pnas.85.20.7795
Abstract: Administration of caused and protein are induced in rat brain after seizures. We a hypothesize that the increase of these putative transcription factor mRNAs dramatic in the brain is part of a programmed genomic response increase of neurons to intense stimulation, which is analogous to the in genomic response of Show more
Authors:
Publication Year: 1957
DOI: DOI not available
Abstract: A comparison that three pyridoxal phosphate enzyme systems in animals convulsed with hydrazides semicarbazide-induced showed inhibition of only l -glutamic acid decarboxylase. seizures are associated with a decreased level of of gamma-aminobutyric acid; pentylenetetrazol seizures were not accompanied by changes in free amino acid levels. Hydrazides have been shown equipotent in Show more
Authors:
Publication Year: 2001
DOI: https://doi.org/10.1523/jneurosci.21-15-05535.2001
Abstract: Retigabine [D-23129; anticonvulsant by KCNQ2/3 channels (Wang et al., 1998)]. Retigabine produced a compound hyperpolarizing shift of the activation curves for KCNQ2/3, KCNQ2, KCNQ3, that and KCNQ4 currents with differential potencies in the following order: is KCNQ3 > KCNQ2/3 > KCNQ2 > KCNQ4, as measured either now by the maximum Show more
Authors:
Publication Year: 2000
DOI: https://doi.org/10.1124/mol.58.3.591
Abstract: Retigabine [N-(2-amino-4-[fluorobenzylamino]-phenyl) to cells. Retigabine-induced currents in CHO-KCNQ2/Q3 cells were inhibited by 60.6 currently ± 11% (n = 4) by the KCNQ2/Q3 blocker, linopirdine available (10 μM), and 82.7 ± 5.4% (n = 4) by antiepileptic BaCl2 (10 mM). The mechanism by which retigabine enhanced KCNQ2/Q3 agents, currents involved large, Show more
Authors:
Publication Year: 2012
DOI: https://doi.org/10.1016/j.seizure.2012.03.001
Abstract: Not available
Authors:
Publication Year: 2012
DOI: https://doi.org/10.1242/dmm.010090
Abstract: Summary The opportunities compounds, which included compounds with known and newly identified anticonvulsant to properties, revealed that they exhibited concentration-dependent inhibition of both locomotor identify activity and PTZ-induced fos transcription, confirming their anticonvulsant characteristics. We novel conclude that this in situ hybridisation assay for fos transcription anticonvulsants in the zebrafish Show more
Authors:
Publication Year: 1998
DOI: https://doi.org/10.1016/s0140-6736(05)79321-1
Abstract: Not available
Authors:
Publication Year: 1978
DOI: https://doi.org/10.1016/0014-2999(78)90369-2
Abstract: Not available
Authors:
Publication Year: 1989
DOI: https://doi.org/10.1213/00000539-198912000-00019
Abstract: This study death bupivacaine, and 4.88 +/- 0.47 mg/kg and 11.4 +/- 0.9 of micrograms/mL for ropivacaine. The margin of safety between the convulsive convulsant and lethal doses was determined by administering two times the and convulsive dose 24 h later. Two dogs given lidocaine died supraconvulsant because of progressive Show more
Authors:
Publication Year: 1995
DOI: https://doi.org/10.1128/aac.39.1.232
Abstract: The epileptogenic their was the most powerful epileptogenic compound among the drugs tested. intracerebroventricular It was approximately three times more potent than benzylpenicillin in administration generating a response and much more potent than other cephalosporins, in such as ceftriaxone, cefoperazone, and cefamandole. No epileptogenic signs were rats. observed with equimolar Show more
Authors:
Publication Year: 1992
DOI: https://doi.org/10.1016/0014-2999(92)90855-x
Abstract: Not available
Authors:
Publication Year: 2014
DOI: https://doi.org/10.1113/jphysiol.2014.277574
Abstract: Sudden unexpected of seizure threshold and increased seizure-induced mortality. Breathing ceased during most death seizures without recovery, whereas cardiac activity persisted for up to in 9 min before terminal arrest. The mortality rate of mice patients of both genotypes was reduced by mechanical ventilation during the with seizure or 5-HT2A Show more
Authors:
Publication Year: 1990
DOI: https://doi.org/10.1111/j.1476-5381.1990.tb14095.x
Abstract: Succinimide derivatives (ethosuximide than ES (IC 50 of 1100 vs 200 μ m (ES); ) but greater in efficacy (100% maximal reduction vs 40% α‐methyl‐α‐phenylsuccinimide for ES). PTZ had no effect on calcium currents, and (MPS)). TMS only reduced LTCC at very high concentrations, and did ES, not occlude MPS Show more
Authors:
Publication Year: 1967
DOI: https://doi.org/10.1111/j.1471-4159.1967.tb09563.x
Abstract: Not available
Authors:
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